| Predicate | Object |
|---|---|
| rdf:type | |
| entrezgene:geneRifText |
Loss of Foxp1/4 in the developing lung and in postnatal secretory epithelium leads to ectopic activation of the goblet cell fate program, in part, through de-repression of the protein disulfide isomerase anterior gradient 2 (Agr2).
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